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Western blotAntibody data
- Antibody Data
- Antigen structure
- References [9]
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- Product number
- 2772 - Provider product page
- Provider
- Cell Signaling Technology, Inc
- Proper citation
- Cell Signaling Technology Cat#2772, RRID:AB_10695870
- Product name
- Bax Antibody
- Antibody type
- Polyclonal
- Reactivity
- Human, Mouse, Rat, Simian
- Host
- Rabbit
- Storage
- -20°C
Submitted references Bcl-2 family genetic profiling reveals microenvironment-specific determinants of chemotherapeutic response.
Mortalin-p53 interaction in cancer cells is stress dependent and constitutes a selective target for cancer therapy.
TWEAK induces apoptosis through a death-signaling complex comprising receptor-interacting protein 1 (RIP1), Fas-associated death domain (FADD), and caspase-8.
Suppression of p38 MAPK and JNK via Akt-mediated inhibition of apoptosis signal-regulating kinase 1 constitutes a core component of the beta-cell pro-survival effects of glucose-dependent insulinotropic polypeptide.
Mitochondrial p53 activates Bak and causes disruption of a Bak-Mcl1 complex.
AKT/protein kinase B regulation of BCL family members during oxysterol-induced apoptosis.
Induction of apoptosis by apicidin, a histone deacetylase inhibitor, via the activation of mitochondria-dependent caspase cascades in human Bcr-Abl-positive leukemia cells.
Glucagon-like peptide-2 receptor activation engages bad and glycogen synthase kinase-3 in a protein kinase A-dependent manner and prevents apoptosis following inhibition of phosphatidylinositol 3-kinase.
Reactive oxygen species-induced phosphorylation of p53 on serine 20 is mediated in part by polo-like kinase-3.
Pritchard JR, Gilbert LA, Meacham CE, Ricks JL, Jiang H, Lauffenburger DA, Hemann MT
Cancer research 2011 Sep 1;71(17):5850-8
Cancer research 2011 Sep 1;71(17):5850-8
Mortalin-p53 interaction in cancer cells is stress dependent and constitutes a selective target for cancer therapy.
Lu WJ, Lee NP, Kaul SC, Lan F, Poon RT, Wadhwa R, Luk JM
Cell death and differentiation 2011 Jun;18(6):1046-56
Cell death and differentiation 2011 Jun;18(6):1046-56
TWEAK induces apoptosis through a death-signaling complex comprising receptor-interacting protein 1 (RIP1), Fas-associated death domain (FADD), and caspase-8.
Ikner A, Ashkenazi A
The Journal of biological chemistry 2011 Jun 17;286(24):21546-54
The Journal of biological chemistry 2011 Jun 17;286(24):21546-54
Suppression of p38 MAPK and JNK via Akt-mediated inhibition of apoptosis signal-regulating kinase 1 constitutes a core component of the beta-cell pro-survival effects of glucose-dependent insulinotropic polypeptide.
Widenmaier SB, Ao Z, Kim SJ, Warnock G, McIntosh CH
The Journal of biological chemistry 2009 Oct 30;284(44):30372-82
The Journal of biological chemistry 2009 Oct 30;284(44):30372-82
Mitochondrial p53 activates Bak and causes disruption of a Bak-Mcl1 complex.
Leu JI, Dumont P, Hafey M, Murphy ME, George DL
Nature cell biology 2004 May;6(5):443-50
Nature cell biology 2004 May;6(5):443-50
AKT/protein kinase B regulation of BCL family members during oxysterol-induced apoptosis.
Rusiñol AE, Thewke D, Liu J, Freeman N, Panini SR, Sinensky MS
The Journal of biological chemistry 2004 Jan 9;279(2):1392-9
The Journal of biological chemistry 2004 Jan 9;279(2):1392-9
Induction of apoptosis by apicidin, a histone deacetylase inhibitor, via the activation of mitochondria-dependent caspase cascades in human Bcr-Abl-positive leukemia cells.
Cheong JW, Chong SY, Kim JY, Eom JI, Jeung HK, Maeng HY, Lee ST, Min YH
Clinical cancer research : an official journal of the American Association for Cancer Research 2003 Oct 15;9(13):5018-27
Clinical cancer research : an official journal of the American Association for Cancer Research 2003 Oct 15;9(13):5018-27
Glucagon-like peptide-2 receptor activation engages bad and glycogen synthase kinase-3 in a protein kinase A-dependent manner and prevents apoptosis following inhibition of phosphatidylinositol 3-kinase.
Yusta B, Estall J, Drucker DJ
The Journal of biological chemistry 2002 Jul 12;277(28):24896-906
The Journal of biological chemistry 2002 Jul 12;277(28):24896-906
Reactive oxygen species-induced phosphorylation of p53 on serine 20 is mediated in part by polo-like kinase-3.
Xie S, Wang Q, Wu H, Cogswell J, Lu L, Jhanwar-Uniyal M, Dai W
The Journal of biological chemistry 2001 Sep 28;276(39):36194-9
The Journal of biological chemistry 2001 Sep 28;276(39):36194-9
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