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Western blotAntibody data
- Antibody Data
- Antigen structure
- References [9]
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- Product number
- ALX-210-003-R050 - Provider product page
- Provider
- Enzo Life Sciences
- Proper citation
- Enzo Life Sciences Cat#ALX-210-003-R050, RRID:AB_2050802
- Product name
- Bax polyclonal antibody
- Antibody type
- Polyclonal
- Antigen
- Synthetic peptide
- Reactivity
- Human, Mouse, Rat
- Host
- Rabbit
- Vial size
- 50 μl
- Storage
- -80°C
Submitted references Mcl-1 is a key regulator of the ovarian reserve.
Sequential cleavage of poly(ADP-ribose)polymerase and appearance of a small Bax-immunoreactive protein are blocked by Bcl-X(L) and caspase inhibitors during staurosporine-induced dopaminergic neuronal apoptosis.
Bax cleavage is mediated by calpain during drug-induced apoptosis.
Developmental expression patterns of Bcl-2, Bcl-x, Bax, and Bak in teeth.
Apoptosis in the terminal endbud of the murine mammary gland: a mechanism of ductal morphogenesis.
Expression of Bcl-2, Bcl-x, and Bax after T cell activation and IL-2 withdrawal.
Upregulation of bax protein levels in neurons following cerebral ischemia.
Overexpression of the Bcl-2 protein increases the half-life of p21Bax.
Immunohistochemical determination of in vivo distribution of Bax, a dominant inhibitor of Bcl-2.
Omari S, Waters M, Naranian T, Kim K, Perumalsamy AL, Chi M, Greenblatt E, Moley KH, Opferman JT, Jurisicova A
Cell death & disease 2015 May 7;6:e1755
Cell death & disease 2015 May 7;6:e1755
Sequential cleavage of poly(ADP-ribose)polymerase and appearance of a small Bax-immunoreactive protein are blocked by Bcl-X(L) and caspase inhibitors during staurosporine-induced dopaminergic neuronal apoptosis.
Kim JE, Oh JH, Choi WS, Chang II, Sohn S, Krajewski S, Reed JC, O'Malley KL, Oh YJ
Journal of neurochemistry 1999 Jun;72(6):2456-63
Journal of neurochemistry 1999 Jun;72(6):2456-63
Bax cleavage is mediated by calpain during drug-induced apoptosis.
Wood DE, Thomas A, Devi LA, Berman Y, Beavis RC, Reed JC, Newcomb EW
Oncogene 1998 Sep 3;17(9):1069-78
Oncogene 1998 Sep 3;17(9):1069-78
Developmental expression patterns of Bcl-2, Bcl-x, Bax, and Bak in teeth.
Krajewski S, Hugger A, Krajewska M, Reed JC, Mai JK
Cell death and differentiation 1998 May;5(5):408-15
Cell death and differentiation 1998 May;5(5):408-15
Apoptosis in the terminal endbud of the murine mammary gland: a mechanism of ductal morphogenesis.
Humphreys RC, Krajewska M, Krnacik S, Jaeger R, Weiher H, Krajewski S, Reed JC, Rosen JM
Development (Cambridge, England) 1996 Dec;122(12):4013-22
Development (Cambridge, England) 1996 Dec;122(12):4013-22
Expression of Bcl-2, Bcl-x, and Bax after T cell activation and IL-2 withdrawal.
Broome HE, Dargan CM, Krajewski S, Reed JC
Journal of immunology (Baltimore, Md. : 1950) 1995 Sep 1;155(5):2311-7
Journal of immunology (Baltimore, Md. : 1950) 1995 Sep 1;155(5):2311-7
Upregulation of bax protein levels in neurons following cerebral ischemia.
Krajewski S, Mai JK, Krajewska M, Sikorska M, Mossakowski MJ, Reed JC
The Journal of neuroscience : the official journal of the Society for Neuroscience 1995 Oct;15(10):6364-76
The Journal of neuroscience : the official journal of the Society for Neuroscience 1995 Oct;15(10):6364-76
Overexpression of the Bcl-2 protein increases the half-life of p21Bax.
Miyashita T, Kitada S, Krajewski S, Horne WA, Delia D, Reed JC
The Journal of biological chemistry 1995 Nov 3;270(44):26049-52
The Journal of biological chemistry 1995 Nov 3;270(44):26049-52
Immunohistochemical determination of in vivo distribution of Bax, a dominant inhibitor of Bcl-2.
Krajewski S, Krajewska M, Shabaik A, Miyashita T, Wang HG, Reed JC
The American journal of pathology 1994 Dec;145(6):1323-36
The American journal of pathology 1994 Dec;145(6):1323-36
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