Antibody data
- Antibody Data
- Antigen structure
- References [3]
- Comments [0]
- Validations [0]
Submit
Validation data
Reference
Comment
Report error
- Product number
- ABIN360473 - Provider product page
- Provider
- antibodies-online
- Product name
- anti-Phosphatidylinositol 4-Kinase Type 2 beta (PI4K2B) (N-Term) antibody
- Antibody type
- Polyclonal
- Description
- Protein G column, eluted with high and low pH buffers and neutralized immediately, followed by dialysis against PBS
- Reactivity
- Human
- Host
- Rabbit
- Epitope
- N-Term
- Vial size
- 0.4 mL
- Storage
- Store the antibody undiluted at 2-8°C for one month or (in aliquots) at-20°C for longer.
- Handling
- Avoid repeated freezing and thawing.
Submitted references Type II phosphatidylinositol 4-kinase beta is a cytosolic and peripheral membrane protein that is recruited to the plasma membrane and activated by Rac-GTP.
Characterization of type II phosphatidylinositol 4-kinase isoforms reveals association of the enzymes with endosomal vesicular compartments.
NCS-1 inhibits insulin-stimulated GLUT4 translocation in 3T3L1 adipocytes through a phosphatidylinositol 4-kinase-dependent pathway.
Wei YJ, Sun HQ, Yamamoto M, Wlodarski P, Kunii K, Martinez M, Barylko B, Albanesi JP, Yin HL
The Journal of biological chemistry 2002 Nov 29;277(48):46586-93
The Journal of biological chemistry 2002 Nov 29;277(48):46586-93
Characterization of type II phosphatidylinositol 4-kinase isoforms reveals association of the enzymes with endosomal vesicular compartments.
Balla A, Tuymetova G, Barshishat M, Geiszt M, Balla T
The Journal of biological chemistry 2002 May 31;277(22):20041-50
The Journal of biological chemistry 2002 May 31;277(22):20041-50
NCS-1 inhibits insulin-stimulated GLUT4 translocation in 3T3L1 adipocytes through a phosphatidylinositol 4-kinase-dependent pathway.
Mora S, Durham PL, Smith JR, Russo AF, Jeromin A, Pessin JE
The Journal of biological chemistry 2002 Jul 26;277(30):27494-500
The Journal of biological chemistry 2002 Jul 26;277(30):27494-500
No comments: Submit comment
No validations: Submit validation data