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- References [10]
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- Product number
- ABIN151306 - Provider product page
- Provider
- antibodies-online
- Product name
- anti-SEC16 Homolog A (S. Cerevisiae) (SEC16A) (AA 600-650) antibody
- Antibody type
- Polyclonal
- Description
- Immunogen affinity purified
- Reactivity
- Human, Rat, Simian
- Host
- Rabbit
- Epitope
- AA 600-650
- Vial size
- 0.1 mL
- Storage
- Store at 4°C. Do not freeze.
- Handling
- Do not freeze.
Submitted references Penta-EF-Hand Protein Peflin Is a Negative Regulator of ER-To-Golgi Transport.
Proteomic profiling of the TRAF3 interactome network reveals a new role for the ER-to-Golgi transport compartments in innate immunity.
Poliovirus infection transiently increases COPII vesicle budding.
Arenavirus infection induces discrete cytosolic structures for RNA replication.
Trafficking-deficient hERG K⁺ channels linked to long QT syndrome are regulated by a microtubule-dependent quality control compartment in the ER.
ERK7 is a negative regulator of protein secretion in response to amino-acid starvation by modulating Sec16 membrane association.
Sec16A defines the site for vesicle budding from the endoplasmic reticulum on exit from mitosis.
Organisation of human ER-exit sites: requirements for the localisation of Sec16 to transitional ER.
Efficient coupling of Sec23-Sec24 to Sec13-Sec31 drives COPII-dependent collagen secretion and is essential for normal craniofacial development.
Palmitoylation and ubiquitination regulate exit of the Wnt signaling protein LRP6 from the endoplasmic reticulum.
Rayl M, Truitt M, Held A, Sargeant J, Thorsen K, Hay JC
PloS one 2016;11(6):e0157227
PloS one 2016;11(6):e0157227
Proteomic profiling of the TRAF3 interactome network reveals a new role for the ER-to-Golgi transport compartments in innate immunity.
van Zuylen WJ, Doyon P, Clément JF, Khan KA, D'Ambrosio LM, Dô F, St-Amant-Verret M, Wissanji T, Emery G, Gingras AC, Meloche S, Servant MJ
PLoS pathogens 2012;8(7):e1002747
PLoS pathogens 2012;8(7):e1002747
Poliovirus infection transiently increases COPII vesicle budding.
Trahey M, Oh HS, Cameron CE, Hay JC
Journal of virology 2012 Sep;86(18):9675-82
Journal of virology 2012 Sep;86(18):9675-82
Arenavirus infection induces discrete cytosolic structures for RNA replication.
Baird NL, York J, Nunberg JH
Journal of virology 2012 Oct;86(20):11301-10
Journal of virology 2012 Oct;86(20):11301-10
Trafficking-deficient hERG K⁺ channels linked to long QT syndrome are regulated by a microtubule-dependent quality control compartment in the ER.
Smith JL, McBride CM, Nataraj PS, Bartos DC, January CT, Delisle BP
American journal of physiology. Cell physiology 2011 Jul;301(1):C75-85
American journal of physiology. Cell physiology 2011 Jul;301(1):C75-85
ERK7 is a negative regulator of protein secretion in response to amino-acid starvation by modulating Sec16 membrane association.
Zacharogianni M, Kondylis V, Tang Y, Farhan H, Xanthakis D, Fuchs F, Boutros M, Rabouille C
The EMBO journal 2011 Aug 16;30(18):3684-700
The EMBO journal 2011 Aug 16;30(18):3684-700
Sec16A defines the site for vesicle budding from the endoplasmic reticulum on exit from mitosis.
Hughes H, Stephens DJ
Journal of cell science 2010 Dec 1;123(Pt 23):4032-8
Journal of cell science 2010 Dec 1;123(Pt 23):4032-8
Organisation of human ER-exit sites: requirements for the localisation of Sec16 to transitional ER.
Hughes H, Budnik A, Schmidt K, Palmer KJ, Mantell J, Noakes C, Johnson A, Carter DA, Verkade P, Watson P, Stephens DJ
Journal of cell science 2009 Aug 15;122(Pt 16):2924-34
Journal of cell science 2009 Aug 15;122(Pt 16):2924-34
Efficient coupling of Sec23-Sec24 to Sec13-Sec31 drives COPII-dependent collagen secretion and is essential for normal craniofacial development.
Townley AK, Feng Y, Schmidt K, Carter DA, Porter R, Verkade P, Stephens DJ
Journal of cell science 2008 Sep 15;121(Pt 18):3025-34
Journal of cell science 2008 Sep 15;121(Pt 18):3025-34
Palmitoylation and ubiquitination regulate exit of the Wnt signaling protein LRP6 from the endoplasmic reticulum.
Abrami L, Kunz B, Iacovache I, van der Goot FG
Proceedings of the National Academy of Sciences of the United States of America 2008 Apr 8;105(14):5384-9
Proceedings of the National Academy of Sciences of the United States of America 2008 Apr 8;105(14):5384-9
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