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- References [6]
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- Immunohistochemistry [1]
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- Product number
- GTX21904 - Provider product page
- Provider
- GeneTex
- Proper citation
- GeneTex Cat#GTX21904, RRID:AB_380314
- Product name
- alpha Synuclein antibody [4B12]
- Antibody type
- Monoclonal
- Reactivity
- Human
- Host
- Mouse
Submitted references Progressive striatonigral degeneration in a transgenic mouse model of multiple system atrophy: translational implications for interventional therapies.
Phosphorylated exogenous alpha-synuclein fibrils exacerbate pathology and induce neuronal dysfunction in mice.
Toll-like receptor 4 stimulation with monophosphoryl lipid A ameliorates motor deficits and nigral neurodegeneration triggered by extraneuronal α-synucleinopathy.
KLK6 proteolysis is implicated in the turnover and uptake of extracellular alpha-synuclein species.
GABA transmission via ATP-dependent K+ channels regulates α-synuclein secretion in mouse striatum.
Brain propagation of transduced α-synuclein involves non-fibrillar protein species and is enhanced in α-synuclein null mice.
Refolo V, Bez F, Polissidis A, Kuzdas-Wood D, Sturm E, Kamaratou M, Poewe W, Stefanis L, Angela Cenci M, Romero-Ramos M, Wenning GK, Stefanova N
Acta neuropathologica communications 2018 Jan 3;6(1):2
Acta neuropathologica communications 2018 Jan 3;6(1):2
Phosphorylated exogenous alpha-synuclein fibrils exacerbate pathology and induce neuronal dysfunction in mice.
Karampetsou M, Ardah MT, Semitekolou M, Polissidis A, Samiotaki M, Kalomoiri M, Majbour N, Xanthou G, El-Agnaf OMA, Vekrellis K
Scientific reports 2017 Nov 28;7(1):16533
Scientific reports 2017 Nov 28;7(1):16533
Toll-like receptor 4 stimulation with monophosphoryl lipid A ameliorates motor deficits and nigral neurodegeneration triggered by extraneuronal α-synucleinopathy.
Venezia S, Refolo V, Polissidis A, Stefanis L, Wenning GK, Stefanova N
Molecular neurodegeneration 2017 Jul 4;12(1):52
Molecular neurodegeneration 2017 Jul 4;12(1):52
KLK6 proteolysis is implicated in the turnover and uptake of extracellular alpha-synuclein species.
Pampalakis G, Sykioti VS, Ximerakis M, Stefanakou-Kalakou I, Melki R, Vekrellis K, Sotiropoulou G
Oncotarget 2017 Feb 28;8(9):14502-14515
Oncotarget 2017 Feb 28;8(9):14502-14515
GABA transmission via ATP-dependent K+ channels regulates α-synuclein secretion in mouse striatum.
Emmanouilidou E, Minakaki G, Keramioti MV, Xylaki M, Balafas E, Chrysanthou-Piterou M, Kloukina I, Vekrellis K
Brain : a journal of neurology 2016 Mar;139(Pt 3):871-90
Brain : a journal of neurology 2016 Mar;139(Pt 3):871-90
Brain propagation of transduced α-synuclein involves non-fibrillar protein species and is enhanced in α-synuclein null mice.
Helwig M, Klinkenberg M, Rusconi R, Musgrove RE, Majbour NK, El-Agnaf OM, Ulusoy A, Di Monte DA
Brain : a journal of neurology 2016 Mar;139(Pt 3):856-70
Brain : a journal of neurology 2016 Mar;139(Pt 3):856-70
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- GeneTex (provider)
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- Experimental details
- IHC analysis of the cortical Lewy body using alpha Synuclein antibody [4B12].
