Antibody data
- Antibody Data
- Antigen structure
- References [7]
- Comments [0]
- Validations [0]
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- Product number
- 26841-1-AP - Provider product page

- Provider
- Proteintech Group
- Product name
- FLVCR1 antibody
- Antibody type
- Polyclonal
- Description
- FLVCR1 antibody (Cat. #26841-1-AP) is a rabbit polyclonal antibody that shows reactivity with human, mouse and has been validated for the following applications: IF, IHC, IP, WB,ELISA.
- Reactivity
- Human, Mouse
- Host
- Rabbit
- Conjugate
- Unconjugated
- Isotype
- IgG
- Vial size
- 20ul, 150ul
Submitted references FLVCR1 Deficiency Impairs Mitochondrial Homeostasis in Retinal Degeneration: Choline as a Potential Therapy.
A Case of Retinopathy-Sensory Neuropathy Syndrome With a Novel Compound Heterozygous FLVCR1 Variant.
The role of FLVCR1 and FLVCR2 in choline transport in the Caco-2 intestinal epithelial cell model and rat small intestine.
Heme allocation in eukaryotic cells relies on mitochondrial heme export through FLVCR1b to cytosolic GAPDH.
Apoptotic metabolites ameliorate bone aging phenotypes via TCOF1/FLVCR1-mediated mitochondrial homeostasis.
Flvcr1a deficiency promotes heme-based energy metabolism dysfunction in skeletal muscle.
Dysregulation of FLVCR1a-dependent mitochondrial calcium handling in neural progenitors causes congenital hydrocephalus.
Wang Y, Li H, Gao Y, He Q, Cai J, Zou R, Zhu X, Zhang L
Clinical & experimental ophthalmology 2026 Jan-Feb;54(1):127-143
Clinical & experimental ophthalmology 2026 Jan-Feb;54(1):127-143
A Case of Retinopathy-Sensory Neuropathy Syndrome With a Novel Compound Heterozygous FLVCR1 Variant.
Nakano Y, Fukui Y, Deguchi K, Matsuoka C, Kawano T, Taira Y, Matsuo A, Osakada Y, Yunoki T, Nomura E, Takemoto M, Morihara R, Yamashita T, Ishiura H
Journal of the peripheral nervous system : JPNS 2025 Dec;30(4):e70082
Journal of the peripheral nervous system : JPNS 2025 Dec;30(4):e70082
The role of FLVCR1 and FLVCR2 in choline transport in the Caco-2 intestinal epithelial cell model and rat small intestine.
Yasujima T, Namba C, Azuma Y, Shinoda Y, Matake I, Yamasaki M, Morimoto H, Namai M, Kusuhara H, Inoue K, Yamashiro T, Yuasa H
Biochimica et biophysica acta. Molecular basis of disease 2025 Aug;1871(6):167883
Biochimica et biophysica acta. Molecular basis of disease 2025 Aug;1871(6):167883
Heme allocation in eukaryotic cells relies on mitochondrial heme export through FLVCR1b to cytosolic GAPDH.
Jayaram DT, Sivaram P, Biswas P, Dai Y, Sweeny EA, Stuehr DJ
Nature communications 2025 Aug 26;16(1):7972
Nature communications 2025 Aug 26;16(1):7972
Apoptotic metabolites ameliorate bone aging phenotypes via TCOF1/FLVCR1-mediated mitochondrial homeostasis.
Qu Y, Meng B, Cai S, Yang B, He Y, Fu C, Li X, Li P, Cao Z, Mao X, Teng W, Shi S
Journal of nanobiotechnology 2024 Sep 6;22(1):549
Journal of nanobiotechnology 2024 Sep 6;22(1):549
Flvcr1a deficiency promotes heme-based energy metabolism dysfunction in skeletal muscle.
Mistretta M, Fiorito V, Allocco AL, Ammirata G, Hsu MY, Digiovanni S, Belicchi M, Napoli L, Ripolone M, Trombetta E, Mauri P, Farini A, Meregalli M, Villa C, Porporato PE, Miniscalco B, Crich SG, Riganti C, Torrente Y, Tolosano E
Cell reports 2024 Mar 26;43(3):113854
Cell reports 2024 Mar 26;43(3):113854
Dysregulation of FLVCR1a-dependent mitochondrial calcium handling in neural progenitors causes congenital hydrocephalus.
Bertino F, Mukherjee D, Bonora M, Bagowski C, Nardelli J, Metani L, Zanin Venturini DI, Chianese D, Santander N, Salaroglio IC, Hentschel A, Quarta E, Genova T, McKinney AA, Allocco AL, Fiorito V, Petrillo S, Ammirata G, De Giorgio F, Dennis E, Allington G, Maier F, Shoukier M, Gloning KP, Munaron L, Mussano F, Salsano E, Pareyson D, di Rocco M, Altruda F, Panagiotakos G, Kahle KT, Gressens P, Riganti C, Pinton PP, Roos A, Arnold T, Tolosano E, Chiabrando D
Cell reports. Medicine 2024 Jul 16;5(7):101647
Cell reports. Medicine 2024 Jul 16;5(7):101647
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