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- Antibody Data
- Antigen structure
- References [4]
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- Product number
- ABIN492998 - Provider product page
- Provider
- antibodies-online
- Product name
- anti-Forkhead Box P3 (FOXP3) (AA 229-250) antibody
- Antibody type
- Polyclonal
- Antigen
- Synthetic peptides selected from forkhead/winged helix transcription factor 3 (Fox3p) corresponding to amino acids 229-250 of mouse protein
- Description
- Affinty chromatography
- Reactivity
- Human, Mouse, Bovine, Simian
- Host
- Rabbit
- Antigen sequence
AQC LLQ REV VQS LEQ QLE LEKE- Epitope
- AA 229-250
- Vial size
- 0.1 mg
- Concentration
- 0.65 - 1.25 mg/mL
- Storage
- Store (in aliquots) at -20°C.
- Handling
- Avoid repeated freezing and thawing.
Submitted references miR-142-3p restricts cAMP production in CD4+CD25- T cells and CD4+CD25+ TREG cells by targeting AC9 mRNA.
FOXP3 inhibits HIV-1 infection of CD4 T-cells via inhibition of LTR transcriptional activity.
Prevalence of Foxp3 positive T regulatory cells is increased during progression of cutaneous squamous tumors.
Disruption of a new forkhead/winged-helix protein, scurfin, results in the fatal lymphoproliferative disorder of the scurfy mouse.
Huang B, Zhao J, Lei Z, Shen S, Li D, Shen GX, Zhang GM, Feng ZH
EMBO reports 2009 Feb;10(2):180-5
EMBO reports 2009 Feb;10(2):180-5
FOXP3 inhibits HIV-1 infection of CD4 T-cells via inhibition of LTR transcriptional activity.
Selliah N, Zhang M, White S, Zoltick P, Sawaya BE, Finkel TH, Cron RQ
Virology 2008 Nov 25;381(2):161-7
Virology 2008 Nov 25;381(2):161-7
Prevalence of Foxp3 positive T regulatory cells is increased during progression of cutaneous squamous tumors.
Jang TJ
Yonsei medical journal 2008 Dec 31;49(6):942-8
Yonsei medical journal 2008 Dec 31;49(6):942-8
Disruption of a new forkhead/winged-helix protein, scurfin, results in the fatal lymphoproliferative disorder of the scurfy mouse.
Brunkow ME, Jeffery EW, Hjerrild KA, Paeper B, Clark LB, Yasayko SA, Wilkinson JE, Galas D, Ziegler SF, Ramsdell F
Nature genetics 2001 Jan;27(1):68-73
Nature genetics 2001 Jan;27(1):68-73
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