Antibody data
- Antibody Data
- Antigen structure
- References [4]
- Comments [0]
- Validations [0]
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- Product number
- ABIN316018 - Provider product page
- Provider
- antibodies-online
- Product name
- anti-Trans-Golgi Network Protein 2 (TGOLN2) antibody
- Antibody type
- Polyclonal
- Description
- IgG purified
- Reactivity
- Human, Mouse, Rat
- Host
- Rabbit
- Vial size
- 0.5 mg
- Storage
- Store at 4°C short term. Aliquot and store at -20°C long term. Avoid freeze-thaw cycles.
- Handling
- Avoid freeze-thaw cycles
Submitted references Characterization of Acyl-CoA synthetase isoforms in pancreatic beta cells: Gene silencing shows participation of ACSL3 and ACSL4 in insulin secretion.
3D electron tomography of brain tissue unveils distinct Golgi structures that sequester cytoplasmic contents in neurons.
Characterization of P4 ATPase Phospholipid Translocases (Flippases) in Human and Rat Pancreatic Beta Cells: THEIR GENE SILENCING INHIBITS INSULIN SECRETION.
The spinal muscular atrophy disease protein SMN is linked to the Golgi network.
Ansari IH, Longacre MJ, Stoker SW, Kendrick MA, O'Neill LM, Zitur LJ, Fernandez LA, Ntambi JM, MacDonald MJ
Archives of biochemistry and biophysics 2017 Mar 15;618:32-43
Archives of biochemistry and biophysics 2017 Mar 15;618:32-43
3D electron tomography of brain tissue unveils distinct Golgi structures that sequester cytoplasmic contents in neurons.
Fernandez-Fernandez MR, Ruiz-Garcia D, Martin-Solana E, Chichon FJ, Carrascosa JL, Fernandez JJ
Journal of cell science 2017 Jan 1;130(1):83-89
Journal of cell science 2017 Jan 1;130(1):83-89
Characterization of P4 ATPase Phospholipid Translocases (Flippases) in Human and Rat Pancreatic Beta Cells: THEIR GENE SILENCING INHIBITS INSULIN SECRETION.
Ansari IU, Longacre MJ, Paulusma CC, Stoker SW, Kendrick MA, MacDonald MJ
The Journal of biological chemistry 2015 Sep 18;290(38):23110-23
The Journal of biological chemistry 2015 Sep 18;290(38):23110-23
The spinal muscular atrophy disease protein SMN is linked to the Golgi network.
Ting CH, Wen HL, Liu HC, Hsieh-Li HM, Li H, Lin-Chao S
PloS one 2012;7(12):e51826
PloS one 2012;7(12):e51826
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