Antibody data
- Antibody Data
- Antigen structure
- References [4]
- Comments [0]
- Validations [0]
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- Product number
- 66651-1-Ig - Provider product page

- Provider
- Proteintech Group
- Product name
- GRASP65,GORASP1 antibody
- Antibody type
- Monoclonal
- Description
- GRASP65,GORASP1 antibody (Cat. #66651-1-Ig) is a mouse monoclonal antibody that shows reactivity with Human, Mouse, Rat and has been validated for the following applications: IHC, WB,ELISA.
- Reactivity
- Human, Mouse, Rat
- Host
- Mouse
- Conjugate
- Unconjugated
- Isotype
- IgG
- Antibody clone number
- 1B2C10
- Vial size
- 20ul, 150ul
Submitted references Discovery of Cell Number-Interstitial Fluid Volume (CIF) Ratio Reveals Secretory Autophagy Pathway to Supply eHsp90α for Wound Healing.
Autophagy-Based Unconventional Secretory for AIM2 Inflammasome Drives DNA Damage Resistance During Intervertebral Disc Degeneration.
Hydrogen peroxide induces Arl1 degradation and impairs Golgi-mediated trafficking.
Golgi apparatus fragmentation participates in oxidized low-density lipoprotein-induced endothelial cell injury.
Chang C, Tang X, Schönthal AH, Chen M, Woodley DT, Wang Y, Liang C, Li W
Cells 2024 Jul 30;13(15)
Cells 2024 Jul 30;13(15)
Autophagy-Based Unconventional Secretory for AIM2 Inflammasome Drives DNA Damage Resistance During Intervertebral Disc Degeneration.
Li S, Liao Z, Luo R, Song Y, Wang K, Feng X, Ou Y, Wu X, Zhang Y, Gao Y, Yin H, Yang C
Frontiers in cell and developmental biology 2021;9:672847
Frontiers in cell and developmental biology 2021;9:672847
Hydrogen peroxide induces Arl1 degradation and impairs Golgi-mediated trafficking.
Ireland SC, Huang H, Zhang J, Li J, Wang Y
Molecular biology of the cell 2020 Aug 1;31(17):1931-1942
Molecular biology of the cell 2020 Aug 1;31(17):1931-1942
Golgi apparatus fragmentation participates in oxidized low-density lipoprotein-induced endothelial cell injury.
Ji G, Song X, Wang L, Li Z, Wu H, Dong H
Journal of cellular biochemistry 2019 Nov;120(11):18862-18870
Journal of cellular biochemistry 2019 Nov;120(11):18862-18870
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