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Western blotAntibody data
- Antibody Data
- Antigen structure
- References [13]
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- Product number
- ABIN113479 - Provider product page
- Provider
- antibodies-online
- Product name
- anti-Nephrosis 1, Congenital, Finnish Type (Nephrin) (NPHS1) (AA 1243-1256), (Intracellular) antibody
- Antibody type
- Polyclonal
- Description
- Serum
- Reactivity
- Human, Mouse
- Host
- Guinea Pig
- Epitope
- AA 1243-1256, Intracellular
- Vial size
- 0.1 mL
- Storage
- Store undiluted at 2-8°C for one month or (in aliquots) at -20°C for longer.
- Handling
- Avoid repeated freezing and thawing.
Submitted references Murine Double Minute-2 Prevents p53-Overactivation-Related Cell Death (Podoptosis) of Podocytes.
Glypican-5 Increases Susceptibility to Nephrotic Damage in Diabetic Kidney.
Marker expression, behaviors, and responses vary in different lines of conditionally immortalized cultured podocytes.
Proteinuria in mice expressing PKB/SGK-resistant GSK3.
Loss of podocyte aPKClambda/iota causes polarity defects and nephrotic syndrome.
Structure-activity relationship of wedelolactone analogues: structural requirements for inhibition of Na+, K+ -ATPase and binding to the central benzodiazepine receptor.
Statins prevent oxidized LDL-induced injury of glomerular podocytes by activating the phosphatidylinositol 3-kinase/AKT-signaling pathway.
Expression of nephrin in acquired human glomerular disease.
Changes in the expression of nephrin gene and protein in experimental diabetic nephropathy.
The murine nephrin gene is specifically expressed in kidney, brain and pancreas: inactivation of the gene leads to massive proteinuria and neonatal death.
Nephrin localizes at the podocyte filtration slit area and is characteristically spliced in the human kidney.
Nephrin is specifically located at the slit diaphragm of glomerular podocytes.
Positionally cloned gene for a novel glomerular protein--nephrin--is mutated in congenital nephrotic syndrome.
Thomasova D, Bruns HA, Kretschmer V, Ebrahim M, Romoli S, Liapis H, Kotb AM, Endlich N, Anders HJ
Journal of the American Society of Nephrology : JASN 2015 Jul;26(7):1513-23
Journal of the American Society of Nephrology : JASN 2015 Jul;26(7):1513-23
Glypican-5 Increases Susceptibility to Nephrotic Damage in Diabetic Kidney.
Okamoto K, Honda K, Doi K, Ishizu T, Katagiri D, Wada T, Tomita K, Ohtake T, Kaneko T, Kobayashi S, Nangaku M, Tokunaga K, Noiri E
The American journal of pathology 2015 Jul;185(7):1889-98
The American journal of pathology 2015 Jul;185(7):1889-98
Marker expression, behaviors, and responses vary in different lines of conditionally immortalized cultured podocytes.
Chittiprol S, Chen P, Petrovic-Djergovic D, Eichler T, Ransom RF
American journal of physiology. Renal physiology 2011 Sep;301(3):F660-71
American journal of physiology. Renal physiology 2011 Sep;301(3):F660-71
Proteinuria in mice expressing PKB/SGK-resistant GSK3.
Boini KM, Amann K, Kempe D, Alessi DR, Lang F
American journal of physiology. Renal physiology 2009 Jan;296(1):F153-9
American journal of physiology. Renal physiology 2009 Jan;296(1):F153-9
Loss of podocyte aPKClambda/iota causes polarity defects and nephrotic syndrome.
Huber TB, Hartleben B, Winkelmann K, Schneider L, Becker JU, Leitges M, Walz G, Haller H, Schiffer M
Journal of the American Society of Nephrology : JASN 2009 Apr;20(4):798-806
Journal of the American Society of Nephrology : JASN 2009 Apr;20(4):798-806
Structure-activity relationship of wedelolactone analogues: structural requirements for inhibition of Na+, K+ -ATPase and binding to the central benzodiazepine receptor.
Pôças ES, Lopes DV, da Silva AJ, Pimenta PH, Leitão FB, Netto CD, Buarque CD, Brito FV, Costa PR, Noël F
Bioorganic & medicinal chemistry 2006 Dec 1;14(23):7962-6
Bioorganic & medicinal chemistry 2006 Dec 1;14(23):7962-6
Statins prevent oxidized LDL-induced injury of glomerular podocytes by activating the phosphatidylinositol 3-kinase/AKT-signaling pathway.
Bussolati B, Deregibus MC, Fonsato V, Doublier S, Spatola T, Procida S, Di Carlo F, Camussi G
Journal of the American Society of Nephrology : JASN 2005 Jul;16(7):1936-47
Journal of the American Society of Nephrology : JASN 2005 Jul;16(7):1936-47
Expression of nephrin in acquired human glomerular disease.
Huh W, Kim DJ, Kim MK, Kim YG, Oh HY, Ruotsalainen V, Tryggvason K
Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association 2002 Mar;17(3):478-84
Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association 2002 Mar;17(3):478-84
Changes in the expression of nephrin gene and protein in experimental diabetic nephropathy.
Aaltonen P, Luimula P, Aström E, Palmen T, Grönholm T, Palojoki E, Jaakkola I, Ahola H, Tikkanen I, Holthöfer H
Laboratory investigation; a journal of technical methods and pathology 2001 Sep;81(9):1185-90
Laboratory investigation; a journal of technical methods and pathology 2001 Sep;81(9):1185-90
The murine nephrin gene is specifically expressed in kidney, brain and pancreas: inactivation of the gene leads to massive proteinuria and neonatal death.
Putaala H, Soininen R, Kilpeläinen P, Wartiovaara J, Tryggvason K
Human molecular genetics 2001 Jan 1;10(1):1-8
Human molecular genetics 2001 Jan 1;10(1):1-8
Nephrin localizes at the podocyte filtration slit area and is characteristically spliced in the human kidney.
Holthöfer H, Ahola H, Solin ML, Wang S, Palmen T, Luimula P, Miettinen A, Kerjaschki D
The American journal of pathology 1999 Nov;155(5):1681-7
The American journal of pathology 1999 Nov;155(5):1681-7
Nephrin is specifically located at the slit diaphragm of glomerular podocytes.
Ruotsalainen V, Ljungberg P, Wartiovaara J, Lenkkeri U, Kestilä M, Jalanko H, Holmberg C, Tryggvason K
Proceedings of the National Academy of Sciences of the United States of America 1999 Jul 6;96(14):7962-7
Proceedings of the National Academy of Sciences of the United States of America 1999 Jul 6;96(14):7962-7
Positionally cloned gene for a novel glomerular protein--nephrin--is mutated in congenital nephrotic syndrome.
Kestilä M, Lenkkeri U, Männikkö M, Lamerdin J, McCready P, Putaala H, Ruotsalainen V, Morita T, Nissinen M, Herva R, Kashtan CE, Peltonen L, Holmberg C, Olsen A, Tryggvason K
Molecular cell 1998 Mar;1(4):575-82
Molecular cell 1998 Mar;1(4):575-82
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