Antibody data
- Antibody Data
- Antigen structure
- References [4]
- Comments [0]
- Validations [0]
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- Product number
- 16694-1-AP - Provider product page
- Provider
- Proteintech Group
- Product name
- MTRF1L antibody
- Antibody type
- Polyclonal
- Description
- MTRF1L antibody (Cat. #16694-1-AP) is a rabbit polyclonal antibody that shows reactivity with human, mouse, rat and has been validated for the following applications: WB,ELISA.
- Reactivity
- Human, Mouse, Rat
- Host
- Rabbit
- Conjugate
- Unconjugated
- Isotype
- IgG
- Vial size
- 20ul, 150ul
Submitted references ZLN005 alleviates PBDE-47 induced impairment of mitochondrial translation and neurotoxicity through PGC-1α/ERRα axis.
Fluoride impairs mitochondrial translation by targeting miR-221-3p/c-Fos/RMND1 axis contributing to neurodevelopment defects.
Muscle denervation reduces mitochondrial biogenesis and mitochondrial translation factor expression in mice.
Exercise-induced mitochondrial biogenesis coincides with the expression of mitochondrial translation factors in murine skeletal muscle.
Tian Z, Li J, Tang H, Liu W, Hou H, Wang C, Li D, Chen G, Xia T, Wang A
Journal of hazardous materials 2024 Jun 5;471:134331
Journal of hazardous materials 2024 Jun 5;471:134331
Fluoride impairs mitochondrial translation by targeting miR-221-3p/c-Fos/RMND1 axis contributing to neurodevelopment defects.
Li D, Zhao Q, Xie L, Wang C, Tian Z, Tang H, Xia T, Wang A
The Science of the total environment 2023 Apr 15;869:161738
The Science of the total environment 2023 Apr 15;869:161738
Muscle denervation reduces mitochondrial biogenesis and mitochondrial translation factor expression in mice.
Yokokawa T, Mori R, Suga T, Isaka T, Hayashi T, Fujita S
Biochemical and biophysical research communications 2020 Jun 18;527(1):146-152
Biochemical and biophysical research communications 2020 Jun 18;527(1):146-152
Exercise-induced mitochondrial biogenesis coincides with the expression of mitochondrial translation factors in murine skeletal muscle.
Yokokawa T, Kido K, Suga T, Isaka T, Hayashi T, Fujita S
Physiological reports 2018 Oct;6(20):e13893
Physiological reports 2018 Oct;6(20):e13893
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