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- References [7]
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- Product number
- 20257-1-AP - Provider product page
- Provider
- Proteintech Group
- Proper citation
- Proteintech Cat#20257-1-AP, RRID:AB_11182704
- Product name
- SCN9A/Nav1.7-Specific antibody
- Antibody type
- Polyclonal
- Description
- SCN9A/Nav1.7-Specific antibody (Cat. #20257-1-AP) is a rabbit polyclonal antibody that shows reactivity with human and has been validated for the following applications: IF, IHC,ELISA.
- Reactivity
- Human
- Host
- Rabbit
- Conjugate
- Unconjugated
- Isotype
- IgG
- Vial size
- 20ul, 150ul
Submitted references Role of macrophages in trigeminal ganglia in ectopic orofacial pain associated with pulpitis.
Ectopic expression of Nav1.7 in spinal dorsal horn neurons induced by NGF contributes to neuropathic pain in a mouse spinal cord injury model.
MicroRNA-96 is required to prevent allodynia by repressing voltage-gated sodium channels in spinal cord.
Connexin 43 contributes to temporomandibular joint inflammation induced-hypernociception via sodium channel 1.7 in trigeminal ganglion.
Glial interleukin-1β upregulates neuronal sodium channel 1.7 in trigeminal ganglion contributing to temporomandibular joint inflammatory hypernociception in rats.
Prostaglandin E(2) Upregulated Trigeminal Ganglionic Sodium Channel 1.7 Involving Temporomandibular Joint Inflammatory Pain in Rats.
Non-steroidal Anti-inflammatory Drugs Attenuate Hyperalgesia and Block Upregulation of Trigeminal Ganglionic Sodium Channel 1.7 after Induction of Temporomandibular Joint Inflammation in Rats.
Sunaga M, Tsuboi Y, Kaizu A, Shinoda M
Journal of oral biosciences 2024 Mar;66(1):145-150
Journal of oral biosciences 2024 Mar;66(1):145-150
Ectopic expression of Nav1.7 in spinal dorsal horn neurons induced by NGF contributes to neuropathic pain in a mouse spinal cord injury model.
Fu Y, Sun L, Zhu F, Xia W, Wen T, Xia R, Yu X, Xu D, Peng C
Frontiers in molecular neuroscience 2023;16:1091096
Frontiers in molecular neuroscience 2023;16:1091096
MicroRNA-96 is required to prevent allodynia by repressing voltage-gated sodium channels in spinal cord.
Sun L, Xia R, Jiang J, Wen T, Huang Z, Qian R, Zhang MD, Zhou M, Peng C
Progress in neurobiology 2021 Jul;202:102024
Progress in neurobiology 2021 Jul;202:102024
Connexin 43 contributes to temporomandibular joint inflammation induced-hypernociception via sodium channel 1.7 in trigeminal ganglion.
Jin YZ, Zhang P, Hao T, Wang LM, Guo MD, Gan YH
Neuroscience letters 2019 Aug 10;707:134301
Neuroscience letters 2019 Aug 10;707:134301
Glial interleukin-1β upregulates neuronal sodium channel 1.7 in trigeminal ganglion contributing to temporomandibular joint inflammatory hypernociception in rats.
Zhang P, Bi RY, Gan YH
Journal of neuroinflammation 2018 Apr 20;15(1):117
Journal of neuroinflammation 2018 Apr 20;15(1):117
Prostaglandin E(2) Upregulated Trigeminal Ganglionic Sodium Channel 1.7 Involving Temporomandibular Joint Inflammatory Pain in Rats.
Zhang P, Gan YH
Inflammation 2017 Jun;40(3):1102-1109
Inflammation 2017 Jun;40(3):1102-1109
Non-steroidal Anti-inflammatory Drugs Attenuate Hyperalgesia and Block Upregulation of Trigeminal Ganglionic Sodium Channel 1.7 after Induction of Temporomandibular Joint Inflammation in Rats.
Bi RY, Ding Y, Gan YH
The Chinese journal of dental research 2016 Mar;19(1):35-42
The Chinese journal of dental research 2016 Mar;19(1):35-42
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Supportive validation
- Submitted by
- Proteintech Group (provider)
- Main image
- Experimental details
- The SCN9A/Nav1.7-Specific antibody from Proteintech is a rabbit polyclonal antibody to a peptide of human SCN9A/Nav1.7-Specific. This antibody recognizes human antigen. The SCN9A/Nav1.7-Specific antibody has been validated for the following applications: ELISA, WB analysis.
