Antibody data
- Antibody Data
- Antigen structure
- References [4]
- Comments [0]
- Validations [0]
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- Product number
- 3024 - Provider product page
- Provider
- Cell Signaling Technology, Inc
- Proper citation
- Cell Signaling Technology Cat#3024, RRID:AB_331253
- Product name
- Phospho-IGF-I Receptor β (Tyr1135/1136)/Insulin Receptor β (Tyr1150/1151) (19H7) Rabbit mAb
- Antibody type
- Monoclonal
- Reactivity
- Human, Mouse, Rat
- Host
- Rabbit
- Storage
- -20°C
Submitted references Phenotype selection reveals coevolution of muscle glycogen and protein and PTEN as a gate keeper for the accretion of muscle mass in adult female mice.
The mechanism of action of the histone deacetylase inhibitor vorinostat involves interaction with the insulin-like growth factor signaling pathway.
Ankrd26 gene disruption enhances adipogenesis of mouse embryonic fibroblasts.
Cell growth, global phosphotyrosine elevation, and c-Met phosphorylation through Src family kinases in colorectal cancer cells.
Sawitzky M, Zeissler A, Langhammer M, Bielohuby M, Stock P, Hammon HM, Görs S, Metges CC, Stoehr BJ, Bidlingmaier M, Fromm-Dornieden C, Baumgartner BG, Christ B, Brenig B, Binder G, Metzger F, Renne U, Hoeflich A
PloS one 2012;7(6):e39711
PloS one 2012;7(6):e39711
The mechanism of action of the histone deacetylase inhibitor vorinostat involves interaction with the insulin-like growth factor signaling pathway.
Sarfstein R, Bruchim I, Fishman A, Werner H
PloS one 2011;6(9):e24468
PloS one 2011;6(9):e24468
Ankrd26 gene disruption enhances adipogenesis of mouse embryonic fibroblasts.
Fei Z, Bera TK, Liu X, Xiang L, Pastan I
The Journal of biological chemistry 2011 Aug 5;286(31):27761-8
The Journal of biological chemistry 2011 Aug 5;286(31):27761-8
Cell growth, global phosphotyrosine elevation, and c-Met phosphorylation through Src family kinases in colorectal cancer cells.
Emaduddin M, Bicknell DC, Bodmer WF, Feller SM
Proceedings of the National Academy of Sciences of the United States of America 2008 Feb 19;105(7):2358-62
Proceedings of the National Academy of Sciences of the United States of America 2008 Feb 19;105(7):2358-62
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