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LearnMAB4247
antibody from R&D Systems
Targeting: CFHR1
CFHL, CFHL1, CFHL1P, CFHR1P, FHR1, H36-1, H36-2, HFL1, HFL2
Western blotAntibody data
- Antibody Data
- Antigen structure
- References [2]
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- Product number
- MAB4247 - Provider product page
- Provider
- R&D Systems
- Product name
- Human Complement Factor H-related 1/CFHR1 Antibody
- Antibody type
- Monoclonal
- Description
- Protein A or G purified from hybridoma culture supernatant. Detects human Complement Factor H-related 1/CFHR1 in direct ELISAs and Western blots. In direct ELISAs and Western blots, no cross-reactivity with recombinant human (rh) CFHR2 or rhCFHR5 is observed.
- Reactivity
- Human
- Host
- Mouse
- Conjugate
- Unconjugated
- Antigen sequence
Q03591- Isotype
- IgG
- Antibody clone number
- 442127
- Vial size
- 100 ug
- Concentration
- LYOPH
- Storage
- Use a manual defrost freezer and avoid repeated freeze-thaw cycles. 12 months from date of receipt, -20 to -70 °C as supplied. 1 month, 2 to 8 °C under sterile conditions after reconstitution. 6 months, -20 to -70 °C under sterile conditions after reconstitution.
Submitted references Complement-Regulatory Proteins CFHR1 and CFHR3 and Patient Response to Anti-CD20 Monoclonal Antibody Therapy.
A novel quantitative hemolytic assay coupled with restriction fragment length polymorphisms analysis enabled early diagnosis of atypical hemolytic uremic syndrome and identified unique predisposing mutations in Japan.
Rogers LM, Mott SL, Smith BJ, Link BK, Sahin D, Weiner GJ
Clinical cancer research : an official journal of the American Association for Cancer Research 2017 Feb 15;23(4):954-961
Clinical cancer research : an official journal of the American Association for Cancer Research 2017 Feb 15;23(4):954-961
A novel quantitative hemolytic assay coupled with restriction fragment length polymorphisms analysis enabled early diagnosis of atypical hemolytic uremic syndrome and identified unique predisposing mutations in Japan.
Yoshida Y, Miyata T, Matsumoto M, Shirotani-Ikejima H, Uchida Y, Ohyama Y, Kokubo T, Fujimura Y
PloS one 2015;10(5):e0124655
PloS one 2015;10(5):e0124655
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