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- Antibody Data
- Antigen structure
- References [6]
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- Validations
- Western blot [1]
- Immunoprecipitation [1]
- Immunohistochemistry [1]
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- Product number
- 13210-1-AP - Provider product page
- Provider
- Proteintech Group
- Proper citation
- Proteintech Cat#13210-1-AP, RRID:AB_2252760
- Product name
- PPP3R1 antibody
- Antibody type
- Polyclonal
- Description
- PPP3R1 antibody (Cat. #13210-1-AP) is a rabbit polyclonal antibody that shows reactivity with human and has been validated for the following applications: IHC, IP, WB,ELISA.
- Reactivity
- Human
- Host
- Rabbit
- Conjugate
- Unconjugated
- Isotype
- IgG
- Vial size
- 20ul, 150ul
Submitted references SMURF1 controls the PPP3/calcineurin complex and TFEB at a regulatory node for lysosomal biogenesis.
Narirutin activates TFEB (transcription factor EB) to protect against Acetaminophen-induced liver injury by targeting PPP3/calcineurin.
Substrate stiffness regulates differentiation of induced pluripotent stem cells into heart valve endothelial cells.
Transcriptional Study Revealed That Boron Supplementation May Alter the Immune-Related Genes Through MAPK Signaling in Ostrich Chick Thymus.
Major contribution of the 3/6/7 class of TRPC channels to myocardial ischemia/reperfusion and cellular hypoxia/reoxygenation injuries.
The calcineurin B subunit induces TNF-related apoptosis-inducing ligand (TRAIL) expression via CD11b-NF-κB pathway in RAW264.7 macrophages.
Xia Q, Zheng H, Li Y, Xu W, Wu C, Xu J, Li S, Zhang L, Dong L
Autophagy 2024 Apr;20(4):735-751
Autophagy 2024 Apr;20(4):735-751
Narirutin activates TFEB (transcription factor EB) to protect against Acetaminophen-induced liver injury by targeting PPP3/calcineurin.
Fang Z, Xu Y, Liu G, Shao Q, Niu X, Tai W, Shen T, Fan M, Chen M, Lei L, Gao W, Song Y, Wang Z, Du X, Li X
Autophagy 2023 Aug;19(8):2240-2256
Autophagy 2023 Aug;19(8):2240-2256
Substrate stiffness regulates differentiation of induced pluripotent stem cells into heart valve endothelial cells.
Cao H, Zhou Q, Liu C, Zhang Y, Xie M, Qiao W, Dong N
Acta biomaterialia 2022 Apr 15;143:115-126
Acta biomaterialia 2022 Apr 15;143:115-126
Transcriptional Study Revealed That Boron Supplementation May Alter the Immune-Related Genes Through MAPK Signaling in Ostrich Chick Thymus.
Xiao K, Yang K, Wang J, Sun P, Huang H, Khaliq H, Naeem MA, Zhong J, Peng K
Biological trace element research 2019 May;189(1):209-223
Biological trace element research 2019 May;189(1):209-223
Major contribution of the 3/6/7 class of TRPC channels to myocardial ischemia/reperfusion and cellular hypoxia/reoxygenation injuries.
He X, Li S, Liu B, Susperreguy S, Formoso K, Yao J, Kang J, Shi A, Birnbaumer L, Liao Y
Proceedings of the National Academy of Sciences of the United States of America 2017 Jun 6;114(23):E4582-E4591
Proceedings of the National Academy of Sciences of the United States of America 2017 Jun 6;114(23):E4582-E4591
The calcineurin B subunit induces TNF-related apoptosis-inducing ligand (TRAIL) expression via CD11b-NF-κB pathway in RAW264.7 macrophages.
Su Z, Xin S, Xu L, Cheng J, Guo J, Li L, Wei Q
Biochemical and biophysical research communications 2012 Jan 13;417(2):777-83
Biochemical and biophysical research communications 2012 Jan 13;417(2):777-83
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Supportive validation
- Submitted by
- Proteintech Group (provider)
- Main image
- Experimental details
- K-562 cells were subjected to SDS PAGE followed by western blot with 13210-1-AP(PPP3R1 antibody) at dilution of 1:400
- Sample type
- cell line
Supportive validation
- Submitted by
- Proteintech Group (provider)
- Main image
- Experimental details
- IP Result of anti-PPP3R1 (IP:13210-1-AP, 3ug; Detection:13210-1-AP 1:300) with HeLa cells lysate 5000ug.
- Sample type
- cell line
Supportive validation
- Submitted by
- Proteintech Group (provider)
- Main image
- Experimental details
- The PPP3R1 antibody from Proteintech is a rabbit polyclonal antibody to a recombinant protein of human PPP3R1. This antibody recognizes human,mouse,rat antigen. The PPP3R1 antibody has been validated for the following applications: ELISA, IHC, IP, WB analysis.
