Antibody data
- Antibody Data
- Antigen structure
- References [4]
- Comments [0]
- Validations
- Western blot [2]
- Immunohistochemistry [2]
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- Product number
- NBP2-15649 - Provider product page
- Provider
- Novus Biologicals
- Product name
- Rabbit Polyclonal Complement Component C3aR Antibody
- Antibody type
- Polyclonal
- Description
- Immunogen affinity purified.
- Reactivity
- Human, Mouse, Rat
- Host
- Rabbit
- Isotype
- IgG
- Vial size
- 0.1 ml
- Storage
- Store at 4C short term. Aliquot and store at -20C long term. Avoid freeze-thaw cycles.
Submitted references Anaphylatoxins enhance Th9 cell recruitment via the CCL20-CCR6 axis in IgA nephropathy.
IL-17A deficiency mitigates bleomycin-induced complement activation during lung fibrosis.
Contribution of the anaphylatoxin receptors, C3aR and C5aR, to the pathogenesis of pulmonary fibrosis.
Crosstalk between TGF-β1 and complement activation augments epithelial injury in pulmonary fibrosis.
Hu X, Feng J, Deng S, Tang J, Liao Z, Luo L, Luo L, Meng T, Gong G, Li X
Journal of nephrology 2020 Feb 8;
Journal of nephrology 2020 Feb 8;
IL-17A deficiency mitigates bleomycin-induced complement activation during lung fibrosis.
Cipolla E, Fisher AJ, Gu H, Mickler EA, Agarwal M, Wilke CA, Kim KK, Moore BB, Vittal R
FASEB journal : official publication of the Federation of American Societies for Experimental Biology 2017 Dec;31(12):5543-5556
FASEB journal : official publication of the Federation of American Societies for Experimental Biology 2017 Dec;31(12):5543-5556
Contribution of the anaphylatoxin receptors, C3aR and C5aR, to the pathogenesis of pulmonary fibrosis.
Gu H, Fisher AJ, Mickler EA, Duerson F 3rd, Cummings OW, Peters-Golden M, Twigg HL 3rd, Woodruff TM, Wilkes DS, Vittal R
FASEB journal : official publication of the Federation of American Societies for Experimental Biology 2016 Jun;30(6):2336-50
FASEB journal : official publication of the Federation of American Societies for Experimental Biology 2016 Jun;30(6):2336-50
Crosstalk between TGF-β1 and complement activation augments epithelial injury in pulmonary fibrosis.
Gu H, Mickler EA, Cummings OW, Sandusky GE, Weber DJ, Gracon A, Woodruff T, Wilkes DS, Vittal R
FASEB journal : official publication of the Federation of American Societies for Experimental Biology 2014 Oct;28(10):4223-34
FASEB journal : official publication of the Federation of American Societies for Experimental Biology 2014 Oct;28(10):4223-34
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Supportive validation
- Submitted by
- Novus Biologicals (provider)
- Main image
- Experimental details
- Western Blot: Complement Component C3a R Antibody [NBP2-15649] - Whole cell extract (30 ug) was separated by 10% SDS-PAGE, and the membrane was blotted with C3a Receptor antibody diluted at 1:1000. The HRP-conjugated anti-rabbit IgG antibody (NBP2-19301) was used to detect the primary antibody.
- Submitted by
- Novus Biologicals (provider)
- Main image
- Experimental details
- Western Blot: Complement Component C3a R Antibody [NBP2-15649] - IL-17A-induced upregulation of C5aR in normal primary human alveolar epithelial cells. This image was submitted via customer Review.
Supportive validation
- Submitted by
- Novus Biologicals (provider)
- Main image
- Experimental details
- Immunohistochemistry-Paraffin: Complement Component C3a R Antibody [NBP2-15649] - Human colon carcinoma, using C3a receptor 1 antibody at 1:500 dilution. Antigen Retrieval: Trilogy™ (EDTA based, pH 8.0) buffer, 15min.
- Submitted by
- Novus Biologicals (provider)
- Main image
- Experimental details
- Immunohistochemistry-Paraffin: Complement Component C3a R Antibody [NBP2-15649] - Tissue staining sections of mouse lung tissue without (left) and with (right) silencing by siRNA. This image was submitted via customer review.