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antibody from antibodies-online
Targeting: FGFR2
BEK, CD332, CEK3, CFD1, ECT1, JWS, K-SAM, KGFR, TK14, TK25
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ImmunocytochemistryAntibody data
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- Antigen structure
- References [5]
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- Validations
- Western blot [3]
- Immunohistochemistry [1]
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- Product number
- ABIN2841762 - Provider product page
- Provider
- antibodies-online
- Product name
- anti-Fibroblast Growth Factor Receptor 2 (FGFR2) (AA 794-821), (C-Term) antibody
- Antibody type
- Polyclonal
- Description
- This antibody is prepared by Saturated Ammonium Sulfate (SAS) precipitation followed by dialysis against PBS.
- Reactivity
- Human
- Host
- Rabbit
- Epitope
- AA 794-821, C-Term
- Antibody clone number
- RB1569
- Vial size
- 80 μL
- Storage
- Maintain refrigerated at 2-8°C for up to 6 months. For long term storage store at -20°C in small aliquots to prevent freeze-thaw cycles.
Submitted references Conditional activation of fibroblast growth factor receptor (FGFR) 1, but not FGFR2, in prostate cancer cells leads to increased osteopontin induction, extracellular signal-regulated kinase activation, and in vivo proliferation.
FGFR2 and WDR11 are neighboring oncogene and tumor suppressor gene on human chromosome 10q26.
Recombination cluster around FGFR2-WDR11-HTPAPL locus on human chromosome 10q26.
P63 alpha mutations lead to aberrant splicing of keratinocyte growth factor receptor in the Hay-Wells syndrome.
Evidence for selective advantage of pathogenic FGFR2 mutations in the male germ line.
Freeman KW, Gangula RD, Welm BE, Ozen M, Foster BA, Rosen JM, Ittmann M, Greenberg NM, Spencer DM
Cancer research 2003 Oct 1;63(19):6237-43
Cancer research 2003 Oct 1;63(19):6237-43
FGFR2 and WDR11 are neighboring oncogene and tumor suppressor gene on human chromosome 10q26.
Katoh M, Katoh M
International journal of oncology 2003 May;22(5):1155-9
International journal of oncology 2003 May;22(5):1155-9
Recombination cluster around FGFR2-WDR11-HTPAPL locus on human chromosome 10q26.
Katoh M, Katoh M
International journal of molecular medicine 2003 May;11(5):579-83
International journal of molecular medicine 2003 May;11(5):579-83
P63 alpha mutations lead to aberrant splicing of keratinocyte growth factor receptor in the Hay-Wells syndrome.
Fomenkov A, Huang YP, Topaloglu O, Brechman A, Osada M, Fomenkova T, Yuriditsky E, Trink B, Sidransky D, Ratovitski E
The Journal of biological chemistry 2003 Jun 27;278(26):23906-14
The Journal of biological chemistry 2003 Jun 27;278(26):23906-14
Evidence for selective advantage of pathogenic FGFR2 mutations in the male germ line.
Goriely A, McVean GA, Röjmyr M, Ingemarsson B, Wilkie AO
Science (New York, N.Y.) 2003 Aug 1;301(5633):643-6
Science (New York, N.Y.) 2003 Aug 1;301(5633):643-6
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Supportive validation
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