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- Antibody Data
- Antigen structure
- References [9]
- Comments [0]
- Validations
- Western blot [1]
- Immunoprecipitation [1]
- Immunohistochemistry [1]
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- Product number
- 11109-1-AP - Provider product page
- Provider
- Proteintech Group
- Proper citation
- Proteintech Cat#11109-1-AP, RRID:AB_2231382
- Product name
- ETFDH antibody
- Antibody type
- Polyclonal
- Description
- ETFDH antibody (Cat. #11109-1-AP) is a rabbit polyclonal antibody that shows reactivity with human, mouse, rat and has been validated for the following applications: IHC, IP, WB,ELISA.
- Reactivity
- Human, Mouse, Rat
- Host
- Rabbit
- Conjugate
- Unconjugated
- Isotype
- IgG
- Vial size
- 20ul, 150ul
Submitted references Propofol Inhibits Proliferation and Augments the Anti-Tumor Effect of Doxorubicin and Paclitaxel Partly Through Promoting Ferroptosis in Triple-Negative Breast Cancer Cells.
Clinical, pathological and genetic features and follow-up of 110 patients with late-onset MADD: a single-center retrospective study.
Mitochondrial FAD shortage in SLC25A32 deficiency affects folate-mediated one-carbon metabolism.
CHIP control degradation of mutant ETF:QO through ubiquitylation in late-onset multiple acyl-CoA dehydrogenase deficiency.
Expression and significance of ETFDH in hepatocellular carcinoma.
ETFDH Mutations and Flavin Adenine Dinucleotide Homeostasis Disturbance Are Essential for Developing Riboflavin-Responsive Multiple Acyl-Coenzyme A Dehydrogenation Deficiency.
Significant clinical heterogeneity with similar ETFDH genotype in three Chinese patients with late-onset multiple acyl-CoA dehydrogenase deficiency.
Molecular analysis of 51 unrelated pedigrees with late-onset multiple acyl-CoA dehydrogenation deficiency (MADD) in southern China confirmed the most common ETFDH mutation and high carrier frequency of c.250G>A.
Riboflavin-responsive lipid-storage myopathy caused by ETFDH gene mutations.
Sun C, Liu P, Pei L, Zhao M, Huang Y
Frontiers in oncology 2022;12:837974
Frontiers in oncology 2022;12:837974
Clinical, pathological and genetic features and follow-up of 110 patients with late-onset MADD: a single-center retrospective study.
Wen B, Tang S, Lv X, Li D, Xu J, Olsen RKJ, Zhao Y, Li W, Wang T, Shao K, Zhao D, Yan C
Human molecular genetics 2022 Mar 31;31(7):1115-1129
Human molecular genetics 2022 Mar 31;31(7):1115-1129
Mitochondrial FAD shortage in SLC25A32 deficiency affects folate-mediated one-carbon metabolism.
Peng MZ, Shao YX, Li XZ, Zhang KD, Cai YN, Lin YT, Jiang MY, Liu ZC, Su XY, Zhang W, Jiang XL, Liu L
Cellular and molecular life sciences : CMLS 2022 Jun 21;79(7):375
Cellular and molecular life sciences : CMLS 2022 Jun 21;79(7):375
CHIP control degradation of mutant ETF:QO through ubiquitylation in late-onset multiple acyl-CoA dehydrogenase deficiency.
Liu XY, Chen XJ, Zhao M, Wang ZQ, Chen HZ, Li HF, Wang CJ, Wu SF, Peng C, Yin Y, Fu HX, Lin MT, Yu L, Xiong ZQ, Wu ZY, Wang N
Journal of inherited metabolic disease 2021 Mar;44(2):450-468
Journal of inherited metabolic disease 2021 Mar;44(2):450-468
Expression and significance of ETFDH in hepatocellular carcinoma.
Wu Y, Zhang X, Shen R, Huang J, Lu X, Zheng G, Chen X
Pathology, research and practice 2019 Dec;215(12):152702
Pathology, research and practice 2019 Dec;215(12):152702
ETFDH Mutations and Flavin Adenine Dinucleotide Homeostasis Disturbance Are Essential for Developing Riboflavin-Responsive Multiple Acyl-Coenzyme A Dehydrogenation Deficiency.
Xu J, Li D, Lv J, Xu X, Wen B, Lin P, Liu F, Ji K, Shan J, Li H, Li W, Zhao Y, Zhao D, Pok JY, Yan C
Annals of neurology 2018 Nov;84(5):659-673
Annals of neurology 2018 Nov;84(5):659-673
Significant clinical heterogeneity with similar ETFDH genotype in three Chinese patients with late-onset multiple acyl-CoA dehydrogenase deficiency.
Fu HX, Liu XY, Wang ZQ, Jin M, Wang DN, He JJ, Lin MT, Wang N
Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology 2016 Jul;37(7):1099-105
Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology 2016 Jul;37(7):1099-105
Molecular analysis of 51 unrelated pedigrees with late-onset multiple acyl-CoA dehydrogenation deficiency (MADD) in southern China confirmed the most common ETFDH mutation and high carrier frequency of c.250G>A.
Wang ZQ, Chen XJ, Murong SX, Wang N, Wu ZY
Journal of molecular medicine (Berlin, Germany) 2011 Jun;89(6):569-76
Journal of molecular medicine (Berlin, Germany) 2011 Jun;89(6):569-76
Riboflavin-responsive lipid-storage myopathy caused by ETFDH gene mutations.
Wen B, Dai T, Li W, Zhao Y, Liu S, Zhang C, Li H, Wu J, Li D, Yan C
Journal of neurology, neurosurgery, and psychiatry 2010 Feb;81(2):231-6
Journal of neurology, neurosurgery, and psychiatry 2010 Feb;81(2):231-6
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Supportive validation
- Submitted by
- Proteintech Group (provider)
- Main image
- Experimental details
- human liver tissue were subjected to SDS PAGE followed by western blot with 11109-1-AP(ETFDH antibody) at dilution of 1:1000
- Sample type
- tissue
Supportive validation
- Submitted by
- Proteintech Group (provider)
- Main image
- Experimental details
- IP Result of anti-ETFDH (IP:11109-1-AP, 3ug; Detection:11109-1-AP 1:500) with mouse brain tissue lysate 8000ug.
- Sample type
- tissue
Supportive validation
- Submitted by
- Proteintech Group (provider)
- Main image
- Experimental details
- The ETFDH antibody from Proteintech is a rabbit polyclonal antibody to a recombinant protein of human ETFDH. This antibody recognizes human,mouse,rat antigen. The ETFDH antibody has been validated for the following applications: ELISA, WB, IHC, IP analysis.
