Antibody data
- Antibody Data
- Antigen structure
- References [4]
- Comments [0]
- Validations [0]
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- Product number
- 2144 - Provider product page
- Provider
- Cell Signaling Technology, Inc
- Proper citation
- Cell Signaling Technology Cat#2144, RRID:AB_2210548
- Product name
- α-Tubulin Antibody
- Antibody type
- Polyclonal
- Reactivity
- Human, Mouse, Rat, Bovine, Drosophila, Simian
- Host
- Rabbit
- Storage
- -20°C
Submitted references Resveratrol inhibits high glucose-induced PI3K/Akt/ERK-dependent interleukin-17 expression in primary mouse cardiac fibroblasts.
Requirement of HDAC6 for transforming growth factor-beta1-induced epithelial-mesenchymal transition.
Deactivation of Akt and STAT3 signaling promotes apoptosis, inhibits proliferation, and enhances the sensitivity of hepatocellular carcinoma cells to an anticancer agent, Atiprimod.
Glucose-dependent insulinotropic polypeptide (GIP) stimulation of pancreatic beta-cell survival is dependent upon phosphatidylinositol 3-kinase (PI3K)/protein kinase B (PKB) signaling, inactivation of the forkhead transcription factor Foxo1, and down-regulation of bax expression.
Venkatachalam K, Mummidi S, Cortez DM, Prabhu SD, Valente AJ, Chandrasekar B
American journal of physiology. Heart and circulatory physiology 2008 May;294(5):H2078-87
American journal of physiology. Heart and circulatory physiology 2008 May;294(5):H2078-87
Requirement of HDAC6 for transforming growth factor-beta1-induced epithelial-mesenchymal transition.
Shan B, Yao TP, Nguyen HT, Zhuo Y, Levy DR, Klingsberg RC, Tao H, Palmer ML, Holder KN, Lasky JA
The Journal of biological chemistry 2008 Jul 25;283(30):21065-73
The Journal of biological chemistry 2008 Jul 25;283(30):21065-73
Deactivation of Akt and STAT3 signaling promotes apoptosis, inhibits proliferation, and enhances the sensitivity of hepatocellular carcinoma cells to an anticancer agent, Atiprimod.
Choudhari SR, Khan MA, Harris G, Picker D, Jacob GS, Block T, Shailubhai K
Molecular cancer therapeutics 2007 Jan;6(1):112-21
Molecular cancer therapeutics 2007 Jan;6(1):112-21
Glucose-dependent insulinotropic polypeptide (GIP) stimulation of pancreatic beta-cell survival is dependent upon phosphatidylinositol 3-kinase (PI3K)/protein kinase B (PKB) signaling, inactivation of the forkhead transcription factor Foxo1, and down-regulation of bax expression.
Kim SJ, Winter K, Nian C, Tsuneoka M, Koda Y, McIntosh CH
The Journal of biological chemistry 2005 Jun 10;280(23):22297-307
The Journal of biological chemistry 2005 Jun 10;280(23):22297-307
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