Antibody data
- Antibody Data
- Antigen structure
- References [4]
- Comments [0]
- Validations [0]
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- Product number
- 25607-1-AP - Provider product page

- Provider
- Proteintech Group
- Product name
- TOMM5 antibody
- Antibody type
- Polyclonal
- Description
- TOMM5 antibody (Cat. #25607-1-AP) is a rabbit polyclonal antibody that shows reactivity with human, mouse and has been validated for the following applications: WB,ELISA.
- Reactivity
- Human, Mouse
- Host
- Rabbit
- Conjugate
- Unconjugated
- Isotype
- IgG
- Vial size
- 20ul, 150ul
Submitted references A unified mechanism for mitochondrial damage sensing in PINK1-Parkin-mediated mitophagy.
SENP6 Maintains Mitochondrial Homeostasis by Regulating Mitochondrial Protein Import Through deSUMOylation of TOM40.
Novel reporter of the PINK1-Parkin mitophagy pathway identifies its damage sensor in the import gate.
TOM5 regulates the mitochondrial membrane potential of alveolar epithelial cells in organizing pneumonia.
Thayer JA, Petersen JD, Huang X, Gruel Budet LM, Hawrot J, Ramos DM, Sekine S, Li Y, Ward ME, Narendra DP
The EMBO journal 2026 Jan;45(1):64-105
The EMBO journal 2026 Jan;45(1):64-105
SENP6 Maintains Mitochondrial Homeostasis by Regulating Mitochondrial Protein Import Through deSUMOylation of TOM40.
Hu L, Li J, Guo H, Su L, Dong P, Huang J, Liu Y, Liu X, Luo Z, Xiong W, Ju Z, Zhou Q, Wang H, Wang W
Advanced science (Weinheim, Baden-Wurttemberg, Germany) 2025 Oct;12(40):e03408
Advanced science (Weinheim, Baden-Wurttemberg, Germany) 2025 Oct;12(40):e03408
Novel reporter of the PINK1-Parkin mitophagy pathway identifies its damage sensor in the import gate.
Thayer JA, Petersen JD, Huang X, Hawrot J, Ramos DM, Sekine S, Li Y, Ward ME, Narendra DP
bioRxiv : the preprint server for biology 2025 Feb 20;
bioRxiv : the preprint server for biology 2025 Feb 20;
TOM5 regulates the mitochondrial membrane potential of alveolar epithelial cells in organizing pneumonia.
Qian Y, Li X, Li X, Zhang X, Yuan Q, Wang Z, Zhang M, Huang M, Ji N
Redox report : communications in free radical research 2024 Dec;29(1):2354625
Redox report : communications in free radical research 2024 Dec;29(1):2354625
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