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- Antibody Data
- Antigen structure
- References [11]
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- Validations
- Western blot [1]
- Immunohistochemistry [1]
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- Product number
- 10773-1-AP - Provider product page
- Provider
- Proteintech Group
- Proper citation
- Proteintech Cat#10773-1-AP, RRID:AB_2237549
- Product name
- PRIM1 antibody
- Antibody type
- Polyclonal
- Description
- PRIM1 antibody (Cat. #10773-1-AP) is a rabbit polyclonal antibody that shows reactivity with human, mouse, rat and has been validated for the following applications: IF, IHC, IP, WB,ELISA.
- Reactivity
- Human, Mouse, Rat
- Host
- Rabbit
- Conjugate
- Unconjugated
- Isotype
- IgG
- Vial size
- 20ul, 150ul
Submitted references Break-induced replication is activated to repair R-loop-associated double-strand breaks in SETX-deficient cells.
53BP1 deficiency leads to hyperrecombination using break-induced replication (BIR).
Targeting Bone Tumours with 45S5 Bioactive Glass.
53BP1 deficiency leads to hyperrecombination using break-induced replication (BIR).
RTF2 controls replication repriming and ribonucleotide excision at the replisome.
RTF2 controls replication repriming and ribonucleotide excision at the replisome.
Expression of BRCA1, BRCA2, RAD51, and other DSB repair factors is regulated by CRL4(WDR70).
53BP1-shieldin-dependent DSB processing in BRCA1-deficient cells requires CST-Polα-primase fill-in synthesis.
CST does not evict elongating telomerase but prevents initiation by ssDNA binding.
Optimization of the chicken chorioallantoic membrane assay as reliable in vivo model for the analysis of osteosarcoma.
A Key Role for the Ubiquitin Ligase UBR4 in Myofiber Hypertrophy in Drosophila and Mice.
Wu T, Li Y, Shi LZ, Wu X
bioRxiv : the preprint server for biology 2025 May 19;
bioRxiv : the preprint server for biology 2025 May 19;
53BP1 deficiency leads to hyperrecombination using break-induced replication (BIR).
Shah SB, Li Y, Li S, Hu Q, Wu T, Shi Y, Nguyen T, Ive I, Shi L, Wang H, Wu X
bioRxiv : the preprint server for biology 2024 Sep 13;
bioRxiv : the preprint server for biology 2024 Sep 13;
Targeting Bone Tumours with 45S5 Bioactive Glass.
Fellenberg J, Losch S, Arango-Ospina M, Hildenbrand N, Tripel E, Deng L, Renkawitz T, Westhauser F, Lehner B, Boccaccini AR
International journal of molecular sciences 2024 Oct 9;25(19)
International journal of molecular sciences 2024 Oct 9;25(19)
53BP1 deficiency leads to hyperrecombination using break-induced replication (BIR).
Shah SB, Li Y, Li S, Hu Q, Wu T, Shi Y, Nguyen T, Ive I, Shi L, Wang H, Wu X
Nature communications 2024 Oct 5;15(1):8648
Nature communications 2024 Oct 5;15(1):8648
RTF2 controls replication repriming and ribonucleotide excision at the replisome.
Conti BA, Ruiz PD, Broton C, Blobel NJ, Kottemann MC, Sridhar S, Lach FP, Wiley TF, Sasi NK, Carroll T, Smogorzewska A
Nature communications 2024 Mar 2;15(1):1943
Nature communications 2024 Mar 2;15(1):1943
RTF2 controls replication repriming and ribonucleotide excision at the replisome.
Conti BA, Ruiz PD, Broton C, Blobel NJ, Kottemann MC, Sridhar S, Lach FP, Wiley T, Sasi NK, Carroll T, Smogorzewska A
bioRxiv : the preprint server for biology 2023 Mar 13;
bioRxiv : the preprint server for biology 2023 Mar 13;
Expression of BRCA1, BRCA2, RAD51, and other DSB repair factors is regulated by CRL4(WDR70).
Mirman Z, Sharma K, Carroll TS, de Lange T
DNA repair 2022 May;113:103320
DNA repair 2022 May;113:103320
53BP1-shieldin-dependent DSB processing in BRCA1-deficient cells requires CST-Polα-primase fill-in synthesis.
Mirman Z, Sasi NK, King A, Chapman JR, de Lange T
Nature cell biology 2022 Jan;24(1):51-61
Nature cell biology 2022 Jan;24(1):51-61
CST does not evict elongating telomerase but prevents initiation by ssDNA binding.
Zaug AJ, Lim CJ, Olson CL, Carilli MT, Goodrich KJ, Wuttke DS, Cech TR
Nucleic acids research 2021 Nov 18;49(20):11653-11665
Nucleic acids research 2021 Nov 18;49(20):11653-11665
Optimization of the chicken chorioallantoic membrane assay as reliable in vivo model for the analysis of osteosarcoma.
Kunz P, Schenker A, Sähr H, Lehner B, Fellenberg J
PloS one 2019;14(4):e0215312
PloS one 2019;14(4):e0215312
A Key Role for the Ubiquitin Ligase UBR4 in Myofiber Hypertrophy in Drosophila and Mice.
Hunt LC, Stover J, Haugen B, Shaw TI, Li Y, Pagala VR, Finkelstein D, Barton ER, Fan Y, Labelle M, Peng J, Demontis F
Cell reports 2019 Jul 30;28(5):1268-1281.e6
Cell reports 2019 Jul 30;28(5):1268-1281.e6
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Supportive validation
- Submitted by
- Proteintech Group (provider)
- Main image
- Experimental details
- K-562 cells were subjected to SDS PAGE followed by western blot with 10773-1-AP(PRIM1 antibody) at dilution of 1:500
- Sample type
- cell line
Supportive validation
- Submitted by
- Proteintech Group (provider)
- Main image
- Experimental details
- The PRIM1 antibody from Proteintech is a rabbit polyclonal antibody to a recombinant protein of human PRIM1. This antibody recognizes human, mouse, rat antigen. The PRIM1 antibody has been validated for the following applications: ELISA, WB, IHC analysis.
