4812
antibody from Cell Signaling Technology, Inc
Targeting: NFKBIA
IkappaBalpha, IKBA, MAD-3, NFKBI
Antibody data
- Antibody Data
- Antigen structure
- References [4]
- Comments [0]
- Validations [0]
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- Product number
- 4812 - Provider product page
- Provider
- Cell Signaling Technology, Inc
- Proper citation
- Cell Signaling Technology Cat#4812, RRID:AB_10694416
- Product name
- IκBα (44D4) Rabbit mAb
- Antibody type
- Monoclonal
- Reactivity
- Human, Mouse, Rat, Hamster, Simian, Other
- Host
- Rabbit
- Storage
- -20°C
Submitted references Specific recognition of linear polyubiquitin by A20 zinc finger 7 is involved in NF-κB regulation.
The Ca2+-dependent phosphatase calcineurin controls the formation of the Carma1-Bcl10-Malt1 complex during T cell receptor-induced NF-kappaB activation.
IL-17 activates the canonical NF-kappaB signaling pathway in autoimmune B cells of BXD2 mice to upregulate the expression of regulators of G-protein signaling 16.
Modulation of TLR signaling by multiple MyD88-interacting partners including leucine-rich repeat Fli-I-interacting proteins.
Tokunaga F, Nishimasu H, Ishitani R, Goto E, Noguchi T, Mio K, Kamei K, Ma A, Iwai K, Nureki O
The EMBO journal 2012 Oct 3;31(19):3856-70
The EMBO journal 2012 Oct 3;31(19):3856-70
The Ca2+-dependent phosphatase calcineurin controls the formation of the Carma1-Bcl10-Malt1 complex during T cell receptor-induced NF-kappaB activation.
Palkowitsch L, Marienfeld U, Brunner C, Eitelhuber A, Krappmann D, Marienfeld RB
The Journal of biological chemistry 2011 Mar 4;286(9):7522-34
The Journal of biological chemistry 2011 Mar 4;286(9):7522-34
IL-17 activates the canonical NF-kappaB signaling pathway in autoimmune B cells of BXD2 mice to upregulate the expression of regulators of G-protein signaling 16.
Xie S, Li J, Wang JH, Wu Q, Yang P, Hsu HC, Smythies LE, Mountz JD
Journal of immunology (Baltimore, Md. : 1950) 2010 Mar 1;184(5):2289-96
Journal of immunology (Baltimore, Md. : 1950) 2010 Mar 1;184(5):2289-96
Modulation of TLR signaling by multiple MyD88-interacting partners including leucine-rich repeat Fli-I-interacting proteins.
Dai P, Jeong SY, Yu Y, Leng T, Wu W, Xie L, Chen X
Journal of immunology (Baltimore, Md. : 1950) 2009 Mar 15;182(6):3450-60
Journal of immunology (Baltimore, Md. : 1950) 2009 Mar 15;182(6):3450-60
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