Antibody data
- Antibody Data
- Antigen structure
- References [10]
- Comments [0]
- Validations [0]
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- Product number
- 2344 - Provider product page
- Provider
- Cell Signaling Technology, Inc
- Proper citation
- Cell Signaling Technology Cat#2344, RRID:AB_331488
- Product name
- Phospho-Chk1 (Ser317) Antibody
- Antibody type
- Polyclonal
- Reactivity
- Human, Simian, Other
- Host
- Rabbit
- Storage
- -20°C
Submitted references Persistent activation of DNA damage signaling in response to complex mixtures of PAHs in air particulate matter.
Initiation of genome instability and preneoplastic processes through loss of Fhit expression.
γH2AX, pChk1, and Wip1 as potential markers of persistent DNA damage derived from dibenzo[a,l]pyrene and PAH-containing extracts from contaminated soils.
Stimulus-dependent dynamics of p53 in single cells.
Thr-1989 phosphorylation is a marker of active ataxia telangiectasia-mutated and Rad3-related (ATR) kinase.
Diminished carcinogen detoxification is a novel mechanism for hypoxia-inducible factor 1-mediated genetic instability.
DNA damage-induced cell cycle regulation and function of novel Chk2 phosphoresidues.
UV-induced ataxia-telangiectasia-mutated and Rad3-related (ATR) activation requires replication stress.
Human claspin is required for replication checkpoint control.
An ATR- and Chk1-dependent S checkpoint inhibits replicon initiation following UVC-induced DNA damage.
Jarvis IW, Bergvall C, Bottai M, Westerholm R, Stenius U, Dreij K
Toxicology and applied pharmacology 2013 Feb 1;266(3):408-18
Toxicology and applied pharmacology 2013 Feb 1;266(3):408-18
Initiation of genome instability and preneoplastic processes through loss of Fhit expression.
Saldivar JC, Miuma S, Bene J, Hosseini SA, Shibata H, Sun J, Wheeler LJ, Mathews CK, Huebner K
PLoS genetics 2012;8(11):e1003077
PLoS genetics 2012;8(11):e1003077
γH2AX, pChk1, and Wip1 as potential markers of persistent DNA damage derived from dibenzo[a,l]pyrene and PAH-containing extracts from contaminated soils.
Niziolek-Kierecka M, Dreij K, Lundstedt S, Stenius U
Chemical research in toxicology 2012 Apr 16;25(4):862-72
Chemical research in toxicology 2012 Apr 16;25(4):862-72
Stimulus-dependent dynamics of p53 in single cells.
Batchelor E, Loewer A, Mock C, Lahav G
Molecular systems biology 2011 May 10;7:488
Molecular systems biology 2011 May 10;7:488
Thr-1989 phosphorylation is a marker of active ataxia telangiectasia-mutated and Rad3-related (ATR) kinase.
Nam EA, Zhao R, Glick GG, Bansbach CE, Friedman DB, Cortez D
The Journal of biological chemistry 2011 Aug 19;286(33):28707-14
The Journal of biological chemistry 2011 Aug 19;286(33):28707-14
Diminished carcinogen detoxification is a novel mechanism for hypoxia-inducible factor 1-mediated genetic instability.
Schults MA, Timmermans L, Godschalk RW, Theys J, Wouters BG, van Schooten FJ, Chiu RK
The Journal of biological chemistry 2010 May 7;285(19):14558-64
The Journal of biological chemistry 2010 May 7;285(19):14558-64
DNA damage-induced cell cycle regulation and function of novel Chk2 phosphoresidues.
Buscemi G, Carlessi L, Zannini L, Lisanti S, Fontanella E, Canevari S, Delia D
Molecular and cellular biology 2006 Nov;26(21):7832-45
Molecular and cellular biology 2006 Nov;26(21):7832-45
UV-induced ataxia-telangiectasia-mutated and Rad3-related (ATR) activation requires replication stress.
Ward IM, Minn K, Chen J
The Journal of biological chemistry 2004 Mar 12;279(11):9677-80
The Journal of biological chemistry 2004 Mar 12;279(11):9677-80
Human claspin is required for replication checkpoint control.
Chini CC, Chen J
The Journal of biological chemistry 2003 Aug 8;278(32):30057-62
The Journal of biological chemistry 2003 Aug 8;278(32):30057-62
An ATR- and Chk1-dependent S checkpoint inhibits replicon initiation following UVC-induced DNA damage.
Heffernan TP, Simpson DA, Frank AR, Heinloth AN, Paules RS, Cordeiro-Stone M, Kaufmann WK
Molecular and cellular biology 2002 Dec;22(24):8552-61
Molecular and cellular biology 2002 Dec;22(24):8552-61
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