Antibody data
- Antibody Data
- Antigen structure
- References [4]
- Comments [0]
- Validations [0]
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- Product number
- 9297 - Provider product page
- Provider
- Cell Signaling Technology, Inc
- Proper citation
- Cell Signaling Technology Cat#9297, RRID:AB_2062131
- Product name
- Phospho-Bad (Ser155) Antibody
- Antibody type
- Polyclonal
- Reactivity
- Mouse
- Host
- Rabbit
- Storage
- -20°C
Submitted references Overexpression of copper/zinc superoxide dismutase in transgenic mice protects against neuronal cell death after transient focal ischemia by blocking activation of the Bad cell death signaling pathway.
Glucagon-like peptide-2 receptor activation engages bad and glycogen synthase kinase-3 in a protein kinase A-dependent manner and prevents apoptosis following inhibition of phosphatidylinositol 3-kinase.
14-3-3 inhibits Bad-induced cell death through interaction with serine-136.
BAD Ser-155 phosphorylation regulates BAD/Bcl-XL interaction and cell survival.
Saito A, Hayashi T, Okuno S, Ferrand-Drake M, Chan PH
The Journal of neuroscience : the official journal of the Society for Neuroscience 2003 Mar 1;23(5):1710-8
The Journal of neuroscience : the official journal of the Society for Neuroscience 2003 Mar 1;23(5):1710-8
Glucagon-like peptide-2 receptor activation engages bad and glycogen synthase kinase-3 in a protein kinase A-dependent manner and prevents apoptosis following inhibition of phosphatidylinositol 3-kinase.
Yusta B, Estall J, Drucker DJ
The Journal of biological chemistry 2002 Jul 12;277(28):24896-906
The Journal of biological chemistry 2002 Jul 12;277(28):24896-906
14-3-3 inhibits Bad-induced cell death through interaction with serine-136.
Masters SC, Yang H, Datta SR, Greenberg ME, Fu H
Molecular pharmacology 2001 Dec;60(6):1325-31
Molecular pharmacology 2001 Dec;60(6):1325-31
BAD Ser-155 phosphorylation regulates BAD/Bcl-XL interaction and cell survival.
Tan Y, Demeter MR, Ruan H, Comb MJ
The Journal of biological chemistry 2000 Aug 18;275(33):25865-9
The Journal of biological chemistry 2000 Aug 18;275(33):25865-9
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