Antibody data
- Antibody Data
- Antigen structure
- References [3]
- Comments [0]
- Validations [0]
Submit
Validation data
Reference
Comment
Report error
- Product number
- ABIN362602 - Provider product page
- Provider
- antibodies-online
- Product name
- anti-BCL2-Associated Agonist of Cell Death (BAD) (AA 134-138) antibody
- Antibody type
- Polyclonal
- Antigen
- Peptide sequence around AA 134-138 (S-R-S-A-P) derived from Mouse BAD. Antibodies were produced by immunizing rabbits with synthetic peptide and KLH conjugates.
- Description
- The antibody was affinity-purified from rabbit antiserum by affinity-chromatography using epitope-specific immunogen.
- Reactivity
- Human, Mouse, Rat
- Host
- Rabbit
- Epitope
- AA 134-138
- Isotype
- IgG
- Vial size
- 100 μL
- Concentration
- 1 mg/mL
- Storage
- Store at -20°C for long term preservation (recommended). Store at 4°C for short term use.
Submitted references Inhibition of integrin-linked kinase/protein kinase B/Akt signaling: mechanism for ganglioside-induced apoptosis.
The hepatitis B virus-X protein activates a phosphatidylinositol 3-kinase-dependent survival signaling cascade.
Lipoarabinomannan from Mycobacterium tuberculosis promotes macrophage survival by phosphorylating Bad through a phosphatidylinositol 3-kinase/Akt pathway.
Wang XQ, Sun P, Paller AS
The Journal of biological chemistry 2001 Nov 30;276(48):44504-11
The Journal of biological chemistry 2001 Nov 30;276(48):44504-11
The hepatitis B virus-X protein activates a phosphatidylinositol 3-kinase-dependent survival signaling cascade.
Lee YI, Kang-Park S, Do SI, Lee YI
The Journal of biological chemistry 2001 May 18;276(20):16969-77
The Journal of biological chemistry 2001 May 18;276(20):16969-77
Lipoarabinomannan from Mycobacterium tuberculosis promotes macrophage survival by phosphorylating Bad through a phosphatidylinositol 3-kinase/Akt pathway.
Maiti D, Bhattacharyya A, Basu J
The Journal of biological chemistry 2001 Jan 5;276(1):329-33
The Journal of biological chemistry 2001 Jan 5;276(1):329-33
No comments: Submit comment
No validations: Submit validation data