Antibody data
- Antibody Data
- Antigen structure
- References [4]
- Comments [0]
- Validations [0]
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- Product number
- 66559-1-Ig - Provider product page
- Provider
- Proteintech Group
- Product name
- SMAD1 antibody
- Antibody type
- Monoclonal
- Description
- SMAD1 antibody (Cat. #66559-1-Ig) is a mouse monoclonal antibody that shows reactivity with Human, Mouse, Rat and has been validated for the following applications: IHC, WB, ELISA.
- Reactivity
- Human, Mouse, Rat
- Host
- Mouse
- Conjugate
- Unconjugated
- Isotype
- IgG
- Antibody clone number
- 2D10B8
- Vial size
- 20ul, 150ul
Submitted references UBE2C orchestrates bone formation through stabilization of SMAD1/5.
miR-122-5p targets GREM2 to protect against glucocorticoid-induced endothelial damage through the BMP signaling pathway.
Neuron-specific enolase promotes stem cell-like characteristics of small-cell lung cancer by downregulating NBL1 and activating the BMP2/Smad/ID1 pathway.
SPG6 supports development of acute myeloid leukemia by regulating BMPR2-Smad-Bcl-2/Bcl-xl signaling.
Zhang H, Du Y, Lu D, Wang X, Li Y, Qing J, Zhang Y, Liu H, Lv L, Zhang X, Liu Y, Zhou Y, Zhang P
Bone 2024 Oct;187:117175
Bone 2024 Oct;187:117175
miR-122-5p targets GREM2 to protect against glucocorticoid-induced endothelial damage through the BMP signaling pathway.
Huang X, Jie S, Li W, Li H, Ni J, Liu C
Molecular and cellular endocrinology 2022 Mar 15;544:111541
Molecular and cellular endocrinology 2022 Mar 15;544:111541
Neuron-specific enolase promotes stem cell-like characteristics of small-cell lung cancer by downregulating NBL1 and activating the BMP2/Smad/ID1 pathway.
Lu L, Zha Z, Zhang P, Wang P, Liu X, Fang X, Weng C, Li B, Mao H, Wang L, Guan M, Wu Y, Xu Z, Liu Z, Liu G
Oncogenesis 2022 Apr 29;11(1):21
Oncogenesis 2022 Apr 29;11(1):21
SPG6 supports development of acute myeloid leukemia by regulating BMPR2-Smad-Bcl-2/Bcl-xl signaling.
Chen J, Li C, Zhan R, Yin Y
Biochemical and biophysical research communications 2018 Jun 18;501(1):220-225
Biochemical and biophysical research communications 2018 Jun 18;501(1):220-225
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