Antibody data
- Antibody Data
- Antigen structure
- References [4]
- Comments [0]
- Validations [0]
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- Product number
- 2042 - Provider product page
- Provider
- Cell Signaling Technology, Inc
- Proper citation
- Cell Signaling Technology Cat#2042, RRID:AB_2214870
- Product name
- XIAP Antibody
- Antibody type
- Polyclonal
- Reactivity
- Human, Mouse, Rat, Simian
- Host
- Rabbit
- Storage
- -20°C
Submitted references Bortezomib sensitizes malignant human glioma cells to TRAIL, mediated by inhibition of the NF-{kappa}B signaling pathway.
XIAP gene expression and function is regulated by autocrine and paracrine TGF-beta signaling.
Simultaneous activation of the intrinsic and extrinsic pathways by histone deacetylase (HDAC) inhibitors and tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) synergistically induces mitochondrial damage and apoptosis in human leukemia cells.
Recruitment of the mitochondrial-dependent apoptotic pathway in amyotrophic lateral sclerosis.
Jane EP, Premkumar DR, Pollack IF
Molecular cancer therapeutics 2011 Jan;10(1):198-208
Molecular cancer therapeutics 2011 Jan;10(1):198-208
XIAP gene expression and function is regulated by autocrine and paracrine TGF-beta signaling.
Van Themsche C, Chaudhry P, Leblanc V, Parent S, Asselin E
Molecular cancer 2010 Aug 16;9:216
Molecular cancer 2010 Aug 16;9:216
Simultaneous activation of the intrinsic and extrinsic pathways by histone deacetylase (HDAC) inhibitors and tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) synergistically induces mitochondrial damage and apoptosis in human leukemia cells.
Rosato RR, Almenara JA, Dai Y, Grant S
Molecular cancer therapeutics 2003 Dec;2(12):1273-84
Molecular cancer therapeutics 2003 Dec;2(12):1273-84
Recruitment of the mitochondrial-dependent apoptotic pathway in amyotrophic lateral sclerosis.
Guégan C, Vila M, Rosoklija G, Hays AP, Przedborski S
The Journal of neuroscience : the official journal of the Society for Neuroscience 2001 Sep 1;21(17):6569-76
The Journal of neuroscience : the official journal of the Society for Neuroscience 2001 Sep 1;21(17):6569-76
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