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Western blotAntibody data
- Antibody Data
- Antigen structure
- References [8]
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- Product number
- ABIN314164 - Provider product page
- Provider
- antibodies-online
- Product name
- anti-Hypoxia Inducible Factor 1, alpha Subunit (Basic Helix-Loop-Helix Transcription Factor) (HIF1A) antibody (Biotin)
- Antibody type
- Polyclonal
- Description
- Immunogen affinity purified
- Reactivity
- Human, Mouse, Rat, Goat, Hamster, Rabbit, Simian
- Host
- Rabbit
- Conjugate
- Biotin
- Vial size
- 0.1 mL
- Storage
- Store at 4°C in the dark.
Submitted references High oxygen prevents fetal lethality due to lack of catecholamines.
HIF1alpha is essential for normal intrauterine differentiation of alveolar epithelium and surfactant production in the newborn lung of mice.
Impaired DNA double-strand break repair contributes to chemoresistance in HIF-1 alpha-deficient mouse embryonic fibroblasts.
Adenomatous polyposis coli-mediated hypersensitivity of mouse embryonic fibroblast cell lines to methylmethane sulfonate treatment: implication of base excision repair pathways.
HIF-1 regulates heritable variation and allele expression phenotypes of the macrophage immune response gene SLC11A1 from a Z-DNA forming microsatellite.
Increased expression of HIF-1alpha, nNOS, and VEGF in the cerebral cortex of anemic rats.
Synergistic up-regulation of vascular endothelial growth factor (VEGF) expression in macrophages by adenosine A2A receptor agonists and endotoxin involves transcriptional regulation via the hypoxia response element in the VEGF promoter.
Hypoxia-inducible factor-1alpha is a key regulator of metastasis in a transgenic model of cancer initiation and progression.
Ream MA, Chandra R, Peavey M, Ray AM, Roffler-Tarlov S, Kim HG, Wetsel WC, Rockman HA, Chikaraishi DM
American journal of physiology. Regulatory, integrative and comparative physiology 2008 Sep;295(3):R942-53
American journal of physiology. Regulatory, integrative and comparative physiology 2008 Sep;295(3):R942-53
HIF1alpha is essential for normal intrauterine differentiation of alveolar epithelium and surfactant production in the newborn lung of mice.
Saini Y, Harkema JR, LaPres JJ
The Journal of biological chemistry 2008 Nov 28;283(48):33650-7
The Journal of biological chemistry 2008 Nov 28;283(48):33650-7
Impaired DNA double-strand break repair contributes to chemoresistance in HIF-1 alpha-deficient mouse embryonic fibroblasts.
Wirthner R, Wrann S, Balamurugan K, Wenger RH, Stiehl DP
Carcinogenesis 2008 Dec;29(12):2306-16
Carcinogenesis 2008 Dec;29(12):2306-16
Adenomatous polyposis coli-mediated hypersensitivity of mouse embryonic fibroblast cell lines to methylmethane sulfonate treatment: implication of base excision repair pathways.
Kundu CN, Balusu R, Jaiswal AS, Narayan S
Carcinogenesis 2007 Oct;28(10):2089-95
Carcinogenesis 2007 Oct;28(10):2089-95
HIF-1 regulates heritable variation and allele expression phenotypes of the macrophage immune response gene SLC11A1 from a Z-DNA forming microsatellite.
Bayele HK, Peyssonnaux C, Giatromanolaki A, Arrais-Silva WW, Mohamed HS, Collins H, Giorgio S, Koukourakis M, Johnson RS, Blackwell JM, Nizet V, Srai SK
Blood 2007 Oct 15;110(8):3039-48
Blood 2007 Oct 15;110(8):3039-48
Increased expression of HIF-1alpha, nNOS, and VEGF in the cerebral cortex of anemic rats.
McLaren AT, Marsden PA, Mazer CD, Baker AJ, Stewart DJ, Tsui AK, Li X, Yucel Y, Robb M, Boyd SR, Liu E, Yu J, Hare GM
American journal of physiology. Regulatory, integrative and comparative physiology 2007 Jan;292(1):R403-14
American journal of physiology. Regulatory, integrative and comparative physiology 2007 Jan;292(1):R403-14
Synergistic up-regulation of vascular endothelial growth factor (VEGF) expression in macrophages by adenosine A2A receptor agonists and endotoxin involves transcriptional regulation via the hypoxia response element in the VEGF promoter.
Ramanathan M, Pinhal-Enfield G, Hao I, Leibovich SJ
Molecular biology of the cell 2007 Jan;18(1):14-23
Molecular biology of the cell 2007 Jan;18(1):14-23
Hypoxia-inducible factor-1alpha is a key regulator of metastasis in a transgenic model of cancer initiation and progression.
Liao D, Corle C, Seagroves TN, Johnson RS
Cancer research 2007 Jan 15;67(2):563-72
Cancer research 2007 Jan 15;67(2):563-72
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